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The compound has been found to operate in the postrema and nucleus of the hindbrain solitary tract in order to both induce satiety and regulate appetite by acting on receptors found in the laterodorsal tegmental nucleus, ventral tegmental area, and the hypothalamus [1]

T-REX coupled with co-immunoprecipitation assays revealed that NCBP1-HNEylation caused no perturbation of the association between NCBP1 and ALYREF, PHAX or PRPF4 (Supplementary Fig

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doi: 10.1038/s41598-020-67383-x 77

We further point out that there are there few patients in this study ( n = 7) who were on insulin, hence it is unclear if the observations described here extend to those in whom insulin insufficiency is severe

Reads were aligned to the mm10 genome using BWA (v.0.7.12) 68