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10.1016/j.jot.2023.02.002 348 TsiampaE.TsiampasK.KapogiannisF

Disease progression of IA is credited to the function of T cell adaptive immunity marked by dysregulated production of Th (T-helper cell) cell cytokines (176, 177)

AOMs enhance weight loss and reduce cardiometabolic events, while maintaining muscle mass

Brain Dev 30:3142 Goldsteins G, Hakosalo V, Jaronen M, Keuters MH, Lehtonen , Koistinaho J (2022) CNS Redox Homeostasis and Dysfunction in Neurodegenerative Diseases.Antioxidants (Basel)11 Asslih S, Damri O, Agam G (2021) Neuroinflammation as a Common Denominator of Complex Diseases (Cancer, Diabetes Type 2, and Neuropsychiatric Disorders).Int J Mol Sci22 Nezu J, Tamai I, Oku A, Ohashi R, Yabuuchi H, Hashimoto N, Nikaido H, Sai Y, Koizumi A, Shoji Y, Takada G, Matsuishi T, Yoshino M, Kato H, Ohura T, Tsujimoto G, Hayakawa J, Shimane M, Tsuji A (1999) Primary systemic carnitine deficiency is caused by mutations in a gene encoding sodium ion-dependent carnitine transporter

That does not automatically mean the peptide is useless, but it does mean the confidence level should remain cautious

However, under the conditions we examined, the activities of AsGGT1 and AsGGT3 to deglutamylate -glutamyl- S -allyl-L-cysteine sulfoxide were too weak to perform further enzymatic characterization