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The NLRP3 inflammasome assembles and cleaves pro-caspase-1 into active caspase-1, which then converts pro-IL-1 into its physiologically active form [64]

Kramer M, Kramer MR, Blau H, Bishara J, Axer-Siegel R, Weinberger D

It is important to note that nNOS activity was increased significantly in ischemic stroke modles, which may lead to neuroexcitotoxicity of glutamate in mesocorticolimbic areas ( Evidence That Hyperglutamatergic State Causes Neuroexcitotoxicity In the late 1960s and early 1970s, it was found that sustained exposure to glutamate and other excitatory amino acids may cause neuronal damage [for review refer to Olney, 1986)

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Consequently, these altered metabolic profiles might modulate the circulating immuno-inflammatory responses that accelerate inflammation in the CNS