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Journal of Toxicology and Environmental Health, Part B: Critical Reviews, 9 (6), 485499

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In addition, it can both inhibit cystine uptake into cells via system x thereby promoting oxidative stress and, as it is a substrate inhibitor, promote L-glu release from cells via the same transporter, which can enhance excitotoxicity through activation of mGluR5 (Liu et al., 2009)

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In mouse models of retinal I/R, Fer-1 administration suppressed ferritin degradation and lipid peroxidation, restrained apoptosis and inflammation of the retina, inhibited RGC death, and protected mouse retinas against I/R stress (Qin et al., 2022

doi: 10.1615/critreveukaryotgeneexpr.2015012376