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Meanwhile, increased mitochondrial GLS expression enhances GLN catabolism by Myc oncogene inhibition of miR-23, which in turn targets GLS [23]

Conclusion In sum, perturbation of Met metabolism may promote the damage process of hepatobiliary injury triggered by viral infections/toxins and/or niche abnormalities, with dysregulated inflammatory and immune responses of the liver, and contribute to the obliteration and eventual fibrosis and atresia of the bile ducts in the initiation/progression of BA

Study of the mechanism of sonodynamic therapy in a rat glioma model

Role of Sirt1 plays in nucleus pulposus cells and intervertebral disc degeneration

Most people notice brighter, more even skin within 48 weeks, especially when treatment is combined with a good skincare routine and antioxidant-rich diet

An antioxidant enzyme therapeutic for sepsis