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6e,f and Supplementary Fig

By targeting these acquired dependencies, it is possible to selectively eliminate KRAS-mutant cancer cells while minimizing effects on normal cells that do not harbor KRAS mutations

Mol Metab 2015;4:51-7

The role of ferroptosis in metabolic diseases

This exploration is pivotal for laying the groundwork for developing potential regulatory small molecules aimed at modulating palmitoylation levels through gene expression regulation, with the dual aim of controlling tumour progression and enhancing the efficacy of therapeutic interventions