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The default location is at the plasma membrane of the cell, and lipid peroxidation is unlikely to occur at other subcellular sites: mitochondria, endoplasmic reticulum, and lysosomes

Evidence of oxidant damage in Huntington's disease: translational strategies using antioxidants

227 NLRP3 promotes PAEC ferroptosis in PAH Ferroptosis, an apoptotic-independent mode of cell demise activated through iron-dependent lipid peroxidation, plays a pivotal role in the development of various inflammation-associated diseases

10.1016/j.jep.2016.08.040 198 YaoJ.WangJ.XuY.GuoQ.SunY.LiuJ.et al (2022)

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Its tumor suppressive effects are likely mediated via several targets, including ALDH (which is overexpressed in cancer stem cells) 29,30 , as well as NPL4 homolog ubiquitin recognition factor, the proteasome, and oxidative stress signaling 31,32,33