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When co-cultured with a human-derived monocyte line with normal G6PD activity, HUVECs rendered G6PD-d via inhibition with 6-AN, dehydroepiandosterone (DHEA), or G6PD knockdown displayed increased monocyte adhesion

The clinical significance of this is unknown, but it is possible that non-angiotensin II sources of aldosterone stimulation (e.g

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Comparative effect of benzanthrone and 3-bromobenzanthrone on hepatic xenobiotic metabolism and anti-oxidative defense system in guinea pigs