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In conclusion, our study uncovers a novel post-translational regulatory mechanism by which LDHB-K58 delactylation promotes gastric cancer metastasis through SLC7A11-mediated GSH synthesis and ferroptosis resistance

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Cancer SLC43A2 alters T cell methionine metabolism and histone methylation

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Notably, NSC-based interventions have also been investigated in the context of tumors arising from dysregulated NSCs programs, such as gliomas, primarily as vehicles for targeted drug delivery (Fawzy et al., 2025)