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As cell mass continues to decrease, the increased metabolic demand, continuous exposure to pro-inflammatory cytokines, and sustained hyperglycemic conditions upregulate mitochondrial and ER stress in cells, which further impair cell function and promote stress-mediated stress (179188)

doi: 10.1016/j.neurobiolaging.2015.09.015

Cancer Med 7(9):4447-4455, 2018

However, as it is becoming more clear that PSSG is an important post-translational modification linked to oxidative stress, recent research has highlighted the glutaredoxin (Grx) system, an antioxidant system capable of reversing PSSG formation

Li, X., Wu, Z., Xue, M

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