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Lipophilic antioxidants prevent lipopolysaccharide-induced mitochondrial dysfunction through mitochondrial biogenesis improvement

1F), while the ROS and ferroptosis stresses were alleviated (Fig

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In NASH patients, hepatic CYP2E1 activity was increased and expression specifically localized to steatotic areas compared to patients with steatosis and healthy controls [103,104,105]

However, we acknowledge that a key limitation of our study is that protein levels and activity were not evaluated to establish causality