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These metabolites, in turn, can feedback into the brain through the bloodstream, immune system signaling, or vagal nerve pathways, thereby influencing central immune responses and the homeostasis of the BBB (173)

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Under pathological conditions, phosphatidylethanolamine, derivatives of PUFAs, may undergo oxidation mediated by lipoxygenases, resulting in the formation of lipid peroxides, which can trigger ferroptosis (Uauy et al., 2000)

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