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F.2007Energy sources for glutamate neurotransmission in the retina: absence of the aspartate/glutamate carrier produces reliance on glycolysis in glia

Diabetic neuropathies: unanswered questions

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Kuramochi cells, which are sensitive to DNA-damaging agents but modestly HR proficient 27 , also showed increased RAD51 foci following treatment with DNA-damaging agents combined with KG or carnitine supplementation (Extended Data Fig

Fecal excretion of total carnitine was less than 1% of the administered dose.10 After attainment of steady state following 4 days of oral administration of levocarnitine tablets (1980 mg q12h) to 15 healthy male volunteers, the mean urinary excretion of levocarnitine during a single dosing interval (12h) was about 9% of the orally administered dose (uncorrected for endogenous urinary excretion)

In fact, fatty acid oxidation metabolites inhibit several glycolytic steps such as glucose transport and phosphorylation, 6-phosphofructo-1-kinase (PFK-1), and pyruvate dehydrogenase (91)