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This bispecific molecule is engineered by conjugating a fully human monoclonal anti-human GIP receptor antagonist antibody to two GLP-1 analogue agonist peptides [159]

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There appears to be an anomalous storage of mitochondrial ATP synthase subunit c that is neither the result of mutation nor enhanced expression of the protein but rather a slower degradation of the mitochondrial ATP synthase in comparison with normal cells [116]

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The study shows that female patients with melanoma had significantly better responses to BRAFMEK-targeted therapy than male patients, and in preclinical melanoma models, elevated AR expression impaired treatment efficacy, while AR inhibition improved responses

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