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Neutrophils in hyperglycemic conditions are triggered to produce S100 calcium-binding protein A8/A9 which binds to the receptors of Kupffer cells to enhance the production of thrombopoietin, which in turn interacts with c-MPL receptor on megakaryocytes and bone marrow progenitor cells to increase the proliferation resulting in reticulated thrombocytosis [39]

Choi, Kim 2014)

Lipuma, J

Of note, such metabolomic alterations can be observed in the R group 12 months after the acute disease