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Deferasirox-dependent iron chelation enhances mitochondrial dysfunction and restores p53 signaling by stabilization of p53 family members in leukemic cells

doi: 10.1046/j.1365-2265.2003.01850.x 29 BednarekJWysockiHSowiskiJ

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Together, these results showed that exposure of MST6-I cells to secretomes from white and beige-like adipocytes promoted a metabolic shift towards increased fatty acid uptake and oxidation, increased mitochondrial number, and reduced cytoplasmic lipid accumulation, indicating enhanced fatty acid consumption

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The temporal relationship between various mutations in the VHL gene and renal tumor progression needs to be examined more critically to unequivocally evaluate the cause-and-effect relationships